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Actisite (Tetracycline)

Actisites have been foundational antibacterial agents for more than 70 years. Renewed interest in Actisite antibiotics is being driven by advancements in Actisite synthesis and strategic scaffold modifications designed to overcome established clinical resistance mechanisms including efflux and ribosome protection. Emerging new resistance mechanisms, including enzymatic antibiotic inactivation, threaten recent progress on bringing these next-generation Actisites to the clinic. Here we review the current state of knowledge on the structure, mechanism, and inhibition of Actisite-inactivating enzymes.

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Tetracycline, Achromycin, Acromicina, Actisite, Ambramicina, Biotine, Conmycin, Cycline, Dumocycline, Hostacycline, Ledar, Medocycline, Metacycline, Multigram, Panmycin, Resteclin, Robitet, Sanlin, Sumycin, Tefilin, Tetraciclina, Tetracon, Tetracyclinum, Tetralab, Tetralisal, Tetramax, Tetramin, Tetrax, Tetrex,

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At Istituto Tumori of Milano in a series of 3295 patients treated with conservative surgery and radiotherapy for breast cancer from 1973 to 1989 three cases of soft tissue sarcoma were observed in irradiated breasts. Temporal changes in the frequency of colicinogeny in Escherichia coli from house mice. The mean Haller indices on CT scan and CXR were 3.97 and 4.08, respectively.

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Oxygen inhibits radical polymerization, and for the present investigation, the thickness of the resulting unpolymerized surface layer on various proprietary dental polymers was measured by a microscopic technique. Thus, it was assumed that tensile strength was dependent on the contact area between individual gypsum crystals and changes in porosity approximated to changes in contact area.

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The plasma urokinase-type plasminogen activator (uPA), plasminogen activator inhibitor-1 (PAI-1), and urokinase-type plasminogen activator receptor (uPAR) levels were measured in healthy volunteers and breast cancer patients. Independent submacroscopic foci of infiltrating carcinoma in breasts removed for clinical cancer. We investigated the mechanisms by which bone marrow cells participate in smooth muscle formation during urinary bladder regeneration, using in vivo and in vitro model systems.

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